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Category: Television technology[Synthesis and cytotoxic activity of 2-amino-5-amino-7-aryl-4(3H)-quinazolinones]. A series of new 2-amino-5-amino-7-aryl-4(3H)-quinazolinones (AQQs) were synthesized by aminolysis of 7-aryl-4(3H)-quinazolinones, and their cytotoxic activity was evaluated. AQQs were more potent than the reference drug, and seven compounds of them were more potent than 5-FU. AQQs-7-trifluoromethyl-4-N-methyl-N-[[(5,6-methylenefuroyl) sulfonyl] amino] phenyl-N(5)-[3-(4-morpholinyl) propionyl] aminomethyl] quinazolin-4(3H)-one (1) and AQQs-7-trifluoromethyl-4-N-methyl-N-[[(5,6-methylenefuroyl) sulfonyl] amino] phenyl-N(5)-[[2-(3,5-dimethyl-1-pyrazolyl)ethyl]amino] methyl] quinazolin-4(3H)-one (11) were more potent than 5-FU.A number of symptoms are associated with pathological arterial wall function. These include local media and adventitial thickening with a decrease in media-to-media and adventitial to media ratios, increased wall rigidity, and reduced wall distensibility. Atherosclerosis is characterized by intimal thickening of the aorta and/or other large blood vessels. It is generally held that the thickening, or intima, is due to deposition of cholesterol and other lipids on the innermost layer of the wall, or intima, and that the inflammation is due to the monocyte migration into the intima and its subsequent transformation into foam cells. Atherosclerosis is known to involve pathological hyper-reactivity of the immune system. This increased immune response has been suggested to be due to an increase in cholesterol content of the cell membranes. Specifically, it has been suggested that human aortic intima contains more membrane cholesterol than human aortic media.We created our world because we're human. And that

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